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Case Report of an ABO-Incompatible Living-Donor Liver Transplant for a Familial Amyloid Polyneuropathy Patient
https://doi.org/10.24517/00061888
https://doi.org/10.24517/000618883cddc30a-4cbc-47cd-97a7-ee0fc8d8a352
名前 / ファイル | ライセンス | アクション |
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HO-PR-NAKANUMA-S-479.pdf (255.8 kB)
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Item type | 学術雑誌論文 / Journal Article(1) | |||||
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公開日 | 2021-05-10 | |||||
タイトル | ||||||
タイトル | Case Report of an ABO-Incompatible Living-Donor Liver Transplant for a Familial Amyloid Polyneuropathy Patient | |||||
言語 | ||||||
言語 | eng | |||||
資源タイプ | ||||||
資源タイプ識別子 | http://purl.org/coar/resource_type/c_6501 | |||||
資源タイプ | journal article | |||||
ID登録 | ||||||
ID登録 | 10.24517/00061888 | |||||
ID登録タイプ | JaLC | |||||
著者 |
Nakanuma, Shinichi
× Nakanuma, Shinichi× Takamura, Hiroyuki× Shoji, Masatoshi× Hayashi, Hironori× Tajima, Hidehiro× Nakagawara, Hisatoshi× Miyashita, Tomoharu× Kitagawa, Hirohisa× Tani, Takashi× Ohta, Tetsuo |
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著者別表示 |
中沼, 伸一
× 中沼, 伸一× 高村, 博之× 林, 泰寛× 田島, 秀浩× 中川原, 寿俊× 宮下, 知治× 北川, 裕久× 太田, 哲生 |
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提供者所属 | ||||||
内容記述タイプ | Other | |||||
内容記述 | 金沢大学附属病院肝胆膵・移植外科 | |||||
書誌情報 |
Experimental and Clinical Transplantation 巻 13, 号 5, p. 479-481, 発行日 2014-09-19 |
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ISSN | ||||||
収録物識別子タイプ | ISSN | |||||
収録物識別子 | 1304-0855 | |||||
DOI | ||||||
関連タイプ | isIdenticalTo | |||||
識別子タイプ | DOI | |||||
関連識別子 | 10.6002/ect.2014.0076 | |||||
PMID | ||||||
関連タイプ | isIdenticalTo | |||||
識別子タイプ | PMID | |||||
関連識別子 | 25247607 | |||||
出版者 | ||||||
出版者 | Baskent University, Publishers / The Middle East Society for Organ Transplantation (MESOT) | |||||
抄録 | ||||||
内容記述タイプ | Abstract | |||||
内容記述 | Liver transplant is a treatment for familial amyloid polyneuropathy. Few cases of ABO-incompatible living-donor liver transplant for familial amyloid polyneuropathy exist. The outcome of an ABO-incompatible living-donor liver transplant has improved recently, using local infusion therapy and rituximab prophylaxis. Here, we describe a successful ABO-incompatible living-donor liver transplant in a patient with familial amyloid polyneuropathy in whom disease progression ceased at 2 years’ follow-up. Additionally, no evidence of acute or chronic rejection, or adverse events of the immuno-suppressive therapy, was seen. As a postoperative complication, fatty changes in the grafted liver because of malnutrition or adverse events of corticosteroids were confirmed by a liver biopsy taken early after transplant. The main cause of malnutrition was considered to be gastrointestinal dysfunction caused by familial amyloid poly-neuropathy. Therefore, before deterioration of digestive function, liver transplants should be considered for familial amyloid polyneuropathy. This case suggests that an ABO-incompatible living-donor liver transplant may provide greater opportunities for familial amyloid polyneuropathy patients. | |||||
権利 | ||||||
権利情報 | Copyright © Baskent University | |||||
著者版フラグ | ||||||
出版タイプ | VoR | |||||
出版タイプResource | http://purl.org/coar/version/c_970fb48d4fbd8a85 | |||||
関連URI | ||||||
識別子タイプ | URI | |||||
関連識別子 | http://www.ectrx.org/home | |||||
関連名称 | http://www.ectrx.org/home | |||||
関連URI | ||||||
識別子タイプ | URI | |||||
関連識別子 | http://ectrx.org/forms/ectrxcontentshow.php?doi_id=10.6002/ect.2014.0076 | |||||
関連名称 | http://ectrx.org/forms/ectrxcontentshow.php?doi_id=10.6002/ect.2014.0076 |