{"created":"2023-07-27T06:28:49.214991+00:00","id":13392,"links":{},"metadata":{"_buckets":{"deposit":"a47ab6fe-3acb-4f57-af4a-77ca2fcaa930"},"_deposit":{"created_by":3,"id":"13392","owners":[3],"pid":{"revision_id":0,"type":"depid","value":"13392"},"status":"published"},"_oai":{"id":"oai:kanazawa-u.repo.nii.ac.jp:00013392","sets":["1132:1133:1134"]},"author_link":["21900","468","336","21901","210"],"item_4_biblio_info_8":{"attribute_name":"書誌情報","attribute_value_mlt":[{"bibliographicIssueDates":{"bibliographicIssueDate":"2004-12-01","bibliographicIssueDateType":"Issued"},"bibliographicIssueNumber":"6","bibliographicPageEnd":"261","bibliographicPageStart":"255","bibliographicVolumeNumber":"25","bibliographic_titles":[{"bibliographic_title":"Biomedical Research"}]}]},"item_4_description_21":{"attribute_name":"抄録","attribute_value_mlt":[{"subitem_description":"TE671 is a cell line obtained from the rhabdomyosarcoma cells. In the present study, we examined [Ca2+]i alteration induced by acetylcholine (ACh) in TE671/RD cells with the special attention to ryanodine receptor (RyR) and inositol 1,4,5-triphosphate receptor (IP3R). The change of [Ca2+]i was mediated by muscarinic type 3 (m3) AChR. Both phosphatidylinositol-specific phospholipase C blocker (U73122) and IP3R blocker (2-APB) inhibited ACh-induced [Ca2+]i elevation, suggesting that IP3 pathway is involved in [Ca2+]i alteration. Ryanodine and FK506 increased ACh-induced [Ca2+]i elevation, which was decreased by RyR blocker (ruthenium red). These results suggest that RyR and FK506 binding protein 12 kDa (FKBP12) complex was involved in [Ca2+]i change, and that TE671/RD cell line has a hybrid characteristic of smooth and skeletal muscles. Copyright © 2005 Biomedical Research Press.","subitem_description_type":"Abstract"}]},"item_4_description_5":{"attribute_name":"提供者所属","attribute_value_mlt":[{"subitem_description":"金沢大学医薬保健研究域医学系","subitem_description_type":"Other"}]},"item_4_publisher_17":{"attribute_name":"出版者","attribute_value_mlt":[{"subitem_publisher":"Biomedical Research"}]},"item_4_relation_12":{"attribute_name":"DOI","attribute_value_mlt":[{"subitem_relation_type":"isIdenticalTo","subitem_relation_type_id":{"subitem_relation_type_id_text":"10.2220/biomedres.25.255","subitem_relation_type_select":"DOI"}}]},"item_4_relation_28":{"attribute_name":"関連URI","attribute_value_mlt":[{"subitem_relation_type_id":{"subitem_relation_type_id_text":"http://dx.doi.org/10.2220/biomedres.25.255","subitem_relation_type_select":"DOI"}}]},"item_4_source_id_11":{"attribute_name":"NCID","attribute_value_mlt":[{"subitem_source_identifier":"AA00110128","subitem_source_identifier_type":"NCID"}]},"item_4_source_id_9":{"attribute_name":"ISSN","attribute_value_mlt":[{"subitem_source_identifier":"0388-6107","subitem_source_identifier_type":"ISSN"}]},"item_4_version_type_25":{"attribute_name":"著者版フラグ","attribute_value_mlt":[{"subitem_version_resource":"http://purl.org/coar/version/c_970fb48d4fbd8a85","subitem_version_type":"VoR"}]},"item_creator":{"attribute_name":"著者","attribute_type":"creator","attribute_value_mlt":[{"creatorNames":[{"creatorName":"Edahiro, Shigeki"}],"nameIdentifiers":[{}]},{"creatorNames":[{"creatorName":"Yoshikawa, Hiroaki"}],"nameIdentifiers":[{},{},{},{}]},{"creatorNames":[{"creatorName":"Iwasa, Kazuo"}],"nameIdentifiers":[{},{},{}]},{"creatorNames":[{"creatorName":"Hashii, Minako"}],"nameIdentifiers":[{},{}]},{"creatorNames":[{"creatorName":"Yamada, Masahito"}],"nameIdentifiers":[{},{},{},{}]}]},"item_files":{"attribute_name":"ファイル情報","attribute_type":"file","attribute_value_mlt":[{"accessrole":"open_date","date":[{"dateType":"Available","dateValue":"2017-10-03"}],"displaytype":"detail","filename":"ME-PR-HASHII-M-255.pdf","filesize":[{"value":"155.3 kB"}],"format":"application/pdf","licensetype":"license_note","mimetype":"application/pdf","url":{"label":"ME-PR-HASHII-M-255.pdf","url":"https://kanazawa-u.repo.nii.ac.jp/record/13392/files/ME-PR-HASHII-M-255.pdf"},"version_id":"99897198-8f3c-4191-9bf4-6deac68ed323"}]},"item_language":{"attribute_name":"言語","attribute_value_mlt":[{"subitem_language":"eng"}]},"item_resource_type":{"attribute_name":"資源タイプ","attribute_value_mlt":[{"resourcetype":"journal article","resourceuri":"http://purl.org/coar/resource_type/c_6501"}]},"item_title":"Cytosolic Ca2+ alteration mediates both ryanodine receptor and IP3 receptor in TE671/RD cells","item_titles":{"attribute_name":"タイトル","attribute_value_mlt":[{"subitem_title":"Cytosolic Ca2+ alteration mediates both ryanodine receptor and IP3 receptor in TE671/RD cells"}]},"item_type_id":"4","owner":"3","path":["1134"],"pubdate":{"attribute_name":"公開日","attribute_value":"2017-10-03"},"publish_date":"2017-10-03","publish_status":"0","recid":"13392","relation_version_is_last":true,"title":["Cytosolic Ca2+ alteration mediates both ryanodine receptor and IP3 receptor in TE671/RD cells"],"weko_creator_id":"3","weko_shared_id":-1},"updated":"2023-07-28T01:05:53.174028+00:00"}