@article{oai:kanazawa-u.repo.nii.ac.jp:00013517,
 author = {Kahara, Toshio and Takamura, Toshinari and Ando, Hitoshi and Sakurai, Masaru and Ota, Tsuguhito and Misaki, Tsuguho and Oba, Sakae and Iguchi, Masahara and Komori, Kazutoshi and Kobayashi, Ken-ichi},
 issue = {6},
 journal = {Internal Medicine},
 month = {Jun},
 note = {A 52-year-old man was admitted to our hospital for diabetic ketoacidosis. On admission, HbA1c was 6.5%, anti-GAD antibody 10.3 U/ml, serum amylase 144 IU/l, lipase 169 U/l and elastase-I 1,000 ng/dl. There were no abdominal symptoms, and abdominal CT showed unremarkable findings. He was treated with intensive insulin therapy. After 1 month, urinary excretion of C-peptide was 3.8 μg/day. Serum pancreatic exocrine enzyme concentrations returned to normal after 3 months. After 10 months, anti-GAD antibody had become negative, but insulin therapy was still needed for glycemic control. This report concerns a case of autoimmune fulminating onset type 1 diabetes.},
 pages = {517--520},
 title = {Fulminating Onset Type 1 Diabetes with Positivity for Anti-GAD Antibody and Elevated Pancreatic Exocrine Enzyme Concentrations},
 volume = {42},
 year = {2003}
}