{"created":"2023-07-27T06:56:18.816292+00:00","id":53124,"links":{},"metadata":{"_buckets":{"deposit":"bc8502b4-e797-41d3-bdf5-1ca23c405614"},"_deposit":{"created_by":18,"id":"53124","owners":[18],"pid":{"revision_id":0,"type":"depid","value":"53124"},"status":"published"},"_oai":{"id":"oai:kanazawa-u.repo.nii.ac.jp:00053124","sets":["2812:2813:4085"]},"author_link":["85505","84783"],"item_9_biblio_info_8":{"attribute_name":"書誌情報","attribute_value_mlt":[{"bibliographicIssueDates":{"bibliographicIssueDate":"2022-06-06","bibliographicIssueDateType":"Issued"},"bibliographicPageStart":"5p.","bibliographicVolumeNumber":"2020-07-30 – 2022-03-31","bibliographic_titles":[{"bibliographic_title":"令和3(2021)年度 科学研究費補助金 挑戦的研究(萌芽) 研究成果報告書"},{"bibliographic_title":"2021 Fiscal Year Final Research Report","bibliographic_titleLang":"en"}]}]},"item_9_creator_33":{"attribute_name":"著者別表示","attribute_type":"creator","attribute_value_mlt":[{"creatorNames":[{"creatorName":"Daikoku, Takiko"}],"nameIdentifiers":[{"nameIdentifier":"85505","nameIdentifierScheme":"WEKO"},{"nameIdentifier":"30767249","nameIdentifierScheme":"e-Rad","nameIdentifierURI":"https://kaken.nii.ac.jp/ja/search/?qm=30767249"}]}]},"item_9_description_21":{"attribute_name":"抄録","attribute_value_mlt":[{"subitem_description":"In the present study, we found that disruption of peripheral circadian rhythms may be involved the initiation and progression of endometrial cancer by regulating angiogenesis through the deletion of the Bmal1 gene in mouse models that spontaneously develops endometrial hyperplasia or endometrial cancer.","subitem_description_type":"Abstract"},{"subitem_description":"本研究では、子宮内膜増殖症あるいは子宮体癌を自然発症するマウスモデルでBmal1遺伝子を欠損させることによって、末梢の概日リズムの乱れが、血管新生を制御して、子宮体癌の発症と進展に関与する可能性を見出した。","subitem_description_type":"Abstract"}]},"item_9_description_22":{"attribute_name":"内容記述","attribute_value_mlt":[{"subitem_description":"研究課題/領域番号:20K21651, 研究期間(年度):2020-07-30 – 2022-03-31","subitem_description_type":"Other"},{"subitem_description":"出典：研究課題「がん自然発症マウスを用いた末梢概日リズム異常によるがん発生・進展誘導機構の解析」課題番号20K21651\n（KAKEN：科学研究費助成事業データベース（国立情報学研究所）） \n（https://kaken.nii.ac.jp/ja/report/KAKENHI-PROJECT-20K21651/20K21651seika/）を加工して作成","subitem_description_type":"Other"}]},"item_9_description_5":{"attribute_name":"提供者所属","attribute_value_mlt":[{"subitem_description":"金沢大学学際科学実験センター","subitem_description_type":"Other"}]},"item_9_identifier_registration":{"attribute_name":"ID登録","attribute_value_mlt":[{"subitem_identifier_reg_text":"10.24517/00059409","subitem_identifier_reg_type":"JaLC"}]},"item_9_relation_28":{"attribute_name":"関連URI","attribute_value_mlt":[{"subitem_relation_name":[{"subitem_relation_name_text":"https://kaken.nii.ac.jp/search/?qm=30767249"}],"subitem_relation_type_id":{"subitem_relation_type_id_text":"https://kaken.nii.ac.jp/search/?qm=30767249","subitem_relation_type_select":"URI"}},{"subitem_relation_name":[{"subitem_relation_name_text":"https://kaken.nii.ac.jp/ja/grant/KAKENHI-PROJECT-20K21651/"}],"subitem_relation_type_id":{"subitem_relation_type_id_text":"https://kaken.nii.ac.jp/ja/grant/KAKENHI-PROJECT-20K21651/","subitem_relation_type_select":"URI"}},{"subitem_relation_name":[{"subitem_relation_name_text":"https://kaken.nii.ac.jp/ja/report/KAKENHI-PROJECT-20K21651/20K21651seika/"}],"subitem_relation_type_id":{"subitem_relation_type_id_text":"https://kaken.nii.ac.jp/ja/report/KAKENHI-PROJECT-20K21651/20K21651seika/","subitem_relation_type_select":"URI"}}]},"item_9_version_type_25":{"attribute_name":"著者版フラグ","attribute_value_mlt":[{"subitem_version_resource":"http://purl.org/coar/version/c_ab4af688f83e57aa","subitem_version_type":"AM"}]},"item_creator":{"attribute_name":"著者","attribute_type":"creator","attribute_value_mlt":[{"creatorNames":[{"creatorName":"大黒, 多希子"}],"nameIdentifiers":[{"nameIdentifier":"84783","nameIdentifierScheme":"WEKO"},{"nameIdentifier":"30767249","nameIdentifierScheme":"e-Rad","nameIdentifierURI":"https://kaken.nii.ac.jp/ja/search/?qm=30767249"}]}]},"item_files":{"attribute_name":"ファイル情報","attribute_type":"file","attribute_value_mlt":[{"accessrole":"open_date","date":[{"dateType":"Available","dateValue":"2023-02-17"}],"displaytype":"detail","filename":"DI-PR-DAIKOKU-T-kaken 2022-5p.pdf","filesize":[{"value":"1.7 MB"}],"format":"application/pdf","licensetype":"license_11","mimetype":"application/pdf","url":{"label":"DI-PR-DAIKOKU-T-kaken 2022-5p.pdf","url":"https://kanazawa-u.repo.nii.ac.jp/record/53124/files/DI-PR-DAIKOKU-T-kaken 2022-5p.pdf"},"version_id":"34096d49-3ca2-4ee3-8976-f228673f6c5f"}]},"item_language":{"attribute_name":"言語","attribute_value_mlt":[{"subitem_language":"jpn"}]},"item_resource_type":{"attribute_name":"資源タイプ","attribute_value_mlt":[{"resourcetype":"research report","resourceuri":"http://purl.org/coar/resource_type/c_18ws"}]},"item_title":"がん自然発症マウスを用いた末梢概日リズム異常によるがん発生・進展誘導機構の解析","item_titles":{"attribute_name":"タイトル","attribute_value_mlt":[{"subitem_title":"がん自然発症マウスを用いた末梢概日リズム異常によるがん発生・進展誘導機構の解析"},{"subitem_title":"Analysis of the mechanisms of induction of cancer initiation/progression by abnormal peripheral circadian rhythm using cancer mouse models","subitem_title_language":"en"}]},"item_type_id":"9","owner":"18","path":["4085"],"pubdate":{"attribute_name":"公開日","attribute_value":"2023-02-17"},"publish_date":"2023-02-17","publish_status":"0","recid":"53124","relation_version_is_last":true,"title":["がん自然発症マウスを用いた末梢概日リズム異常によるがん発生・進展誘導機構の解析"],"weko_creator_id":"18","weko_shared_id":-1},"updated":"2024-07-01T05:18:12.701646+00:00"}